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View More by This Developer. Fig 3 Naturally infected piglets show high anti-ApfA immune responses.

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The infected animals develop disease symptoms such as chronic necrotizing pneumonia, acute fibrinous pneumonia, and pleuritis with high mortality, which leads to large economic losses in the pig farming industry worldwide 3.

Type IV fimbriae are critical to mediating the adhesion of A.

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Taken together, these results indicate that type IV fimbriae are essential for mediating the adhesion of A. Currently, the single-antigen vaccines that have been tested in pigs are not desirable.

Construction of apfA mutant and complementary strains.

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Serological response and cross immunity in pigs. Flagella facilitate escape of Salmonella from oncotic macrophages. Naturally infected piglets show high anti-ApfA immune responses.

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The apfA gene is conserved among A. Among these subunit vaccine candidates, the Apx exotoxins are virulence factors that play a predominant role in the pathogenesis of A.

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Transcriptional profiling of Actinobacillus pleuropneumoniae during the acute phase of a natural infection in pigs. Cover Version 0 Votes. This observation implies that additional virulence factors may exist and be involved in the adhesion of A.

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Enhanced biofilm formation and reduced virulence of Actinobacillus pleuropneumoniae luxS mutant. The expression of apfA is enhanced by contact with host cells.

In contrast, all mice administered a control serum from adjuvant-immunized mice died within 24 h postchallenge.

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Google Scholar Articles by Zhou, Y.

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